MOLECULAR MIMICRY AND TYPE 1 DIABETES

  • Cow’s milk was not designed to be consumed by humans.  
  • Some children do not digest cow’s milk completely, which results in partially digested protein.
  • Impaired GI health (leaky gut) allows passage of these proteins into the bloodstream.  
  • The immune system recognizes these as foreign invaders and produces antibodies.
  • These milk protein fragments look like the pancreatic cells that produce insulin (beta cells).
  • The immune system begins to attack the pancreatic cells, eventually resulting in type-1 diabetes.
  • A 1992 study - blood samples taken from 142 diabetic children and controls who did not have diabetes...
  • 100% of the diabetic children had elevated levels of antibodies to cow’s milk proteins; none of the controls had elevated antibody levels
  • Other studies have confirmed results.

Karjalainen J, et al, “A bovine albumin peptide as a possible trigger of insulin-dependent Diabetes Mellitus.” NEJM. 327(1992):302-307

Akerblom HK, Knip M. “Putative environmental factors and Type 1 diabetes.” Diabetes/Metabolism Revs 14 (1998):31-67

Naik RG and Palmer JP. “Preservation of beta-cell function in Type-1 Diabetes.” Diabetes Rev. 7 (1999):154-182